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In additionto these measures, the primary approach of therapyin subjects with low HDL-CH is to reduceLDL-CH to the target level as per theirLDL-CH risk category or to achieve a total CH:HDL-CH ratio of 3.5, whichever is moreintensive. Wade M et al (2012) MDM2, MDMX and p53 in oncogenesis and cancer therapy.

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Hydroxylation of key proline residues by oneof three prolyl-hydroxylases (PHD1-3) targets it to the E3 ubiquitin ligase pVHL,which subsequently leads to HIF1? degradation by the proteasome [ 25].

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